In essence, the authors note that the loss of AR in LebRb neurons left body mass, glucose, and puberty much the same but aided fertility. The mice had more regular cycles and healthier ovaries, and the authors wrote that this recovery likely came after birth, when AR signals fell silent. Losing these signals blunted some effects of prenatal androgen exposure. So, the study shows that AR in LepRb cells ties excess androgen to the loss of cycles, and that the neural circuits for puberty and for female cyclicity lie apart in the brain.