As seen in Figs.1A-C, AR and LebRb neurons overlapped in the hypothalamus. Deleting the receptor worked, and DHT enlarged the anogenital gab in every group. Lepr signals were absent before birth, so the change came only after life. Later, weight, fat and glucose stayed much the same, as shown in Figs. 2A-E, and metabolism held steady. Puberty came late in androgen-exposed females, with or without the receptor, as seen in Figs. 3A-D; the loss of AR in LebRb cells didn’t alter that delay, yet females without the receptor regained regular cycles and more corpora lutea (Figs. 4A-D, 5A-F). Even so, hormone and gene readings in the pituitary and hypothalamus stayed flat (Figs 6A-I). Deleting AR in LepRb neurons left metabolism steady and restored fertility after prenatal androgen exposure, exactly as the authors proposed